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This ECG is from a middle aged man who presented with chest pain and diaphoresis while exercising. He had a VSD repair at age 6.
This ECG shows sinus tachycardia with RBBB + LPFB. ST elevation in III and aVF with reciprocal ST depression in aVL. ST depression in V2-5. The cause was inferoposterior MI, due to severe triple vessel disease and a culprit 100% circumflex occlusion.
This ECG is from a woman in her 70s who presented with sudden nausea, diaphoresis and brief syncope.
This ECG shows subtle concave ST elevation in inferior leads and V4-6. The angiogram was normal, troponins negative and echo showed no wall motion abnormality. The most likely cause was a normal variant.
This ECG is from a man in his 60s who developed hypokalemia during a complicated hospital admission including GI bleed and anaemia.
This ECG shows inverted T waves and inverted U waves with a very prolonged QT. There is subtle ST elevation in inferior and lateral leads. Troponin was elevated, echo showed regional wall motion abnormalities apical and anteriorly. Angiogram showed severe multivessel disease and an apical LAD occlusion of uncertain age. The most likely cause was either a small infarction or takotsubo.
This ECG is from a man in his 60s who presented with chest pain for 48h with diaphoresis.
This ECG shows inferior ST elevation with reciprocal depression in aVL and I. ST depression in V2-4. Deep Q waves inferior leads and tall R waves V1-3. The cause was a completed infarction with 2 likely culprits (RCA and circumflex).
This ECG is from a man in his 60s who presented with chest pain radiating to the back and nausea/vomiting. He had a history of previous MI and stents.
This ECG shows inferior ST elevation (straightened ST segments) without reciprocal changes, with terminal QRS distortion in inferior leads. The cause was an inferior MI with some posterior involvement.
This ECG is from a man in his 50s who presented with chest pain on a background of known coronary artery disease.
This ECG shows ST elevation with Q waves, high voltage QRS and atypical ST morphology for acute ischemia. On angiography he had three vessel disease but no obvious acute culprit. Serial troponins did not rise and serial ECGs showed no evolution.